Molecular and cellular mechanisms underlying the hepatoprotective role of ghrelin against NAFLD progression
Author:
Funder
Instituto de Salud Carlos III
Publisher
Springer Science and Business Media LLC
Subject
General Medicine,Biochemistry,Physiology
Link
https://link.springer.com/content/pdf/10.1007/s13105-022-00933-1.pdf
Reference208 articles.
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2. Afonso MB, Rodrigues PM, Mateus-Pinheiro M, Simao AL, Gaspar MM, Majdi A, Arretxe E, Alonso C, Santos-Laso A, Jimenez-Aguero R et al (2021) RIPK3 acts as a lipid metabolism regulator contributing to inflammation and carcinogenesis in non-alcoholic fatty liver disease. Gut 70:2359–2372
3. Afonso MB, Islam T, Magusto J, Amorim R, Lenoir V, Simoes R, Teixeira J, Silva LC, Wendum D, Jeru I et al (2022) RIPK3 dampens mitochondrial bioenergetics and lipid droplet dynamics in metabolic liver disease. Hepatology. https://doi.org/10.1002/hep.32756
4. Alharbi S (2022) Exogenous administration of unacylated ghrelin attenuates hepatic steatosis in high-fat diet-fed rats by modulating glucose homeostasis, lipogenesis, oxidative stress, and endoplasmic reticulum stress. Biomed Pharmacother 151:113095
5. Álvarez-Sola G, Uriarte I, Latasa MU, Fernández-Barrena MG, Urtasun R, Elizalde M, Barcena-Varela M, Jimenez M, Chang HC, Barbero R et al (2017) Fibroblast growth factor 15/19 (FGF15/19) protects from diet-induced hepatic steatosis: development of an FGF19-based chimeric molecule to promote fatty liver regeneration. Gut 66:1818–1828
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