CFTR supports cell death through ROS-dependent activation of TMEM16F (anoctamin 6)-Reference-Cited by-同舟云学术

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CFTR supports cell death through ROS-dependent activation of TMEM16F (anoctamin 6)

Published:2017-09-05 Issue:2 Volume:470 Page:305-314
ISSN:0031-6768
Container-title:Pflügers Archiv - European Journal of Physiology
language:en
Short-container-title:Pflugers Arch - Eur J Physiol

Author:

Simões Filipa,Ousingsawat Jiraporn,Wanitchakool Podchanart,Fonseca Ana,Cabrita Inês,Benedetto Roberta,Schreiber Rainer,Kunzelmann Karl

Funder

Deutsche Forschungsgemeinschaft

Cystic Fibrosis Trust

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Clinical Biochemistry,Physiology

Link

http://link.springer.com/article/10.1007/s00424-017-2065-0/fulltext.html

Reference45 articles.

1. Benedetto R, Ousingsawat J, Wanitchakool P, Zhang Y, Holtzman MJ, Amaral M, Rock JR, Schreiber R, Kunzelmann K (2017) Epithelial chloride transport by CFTR requires TMEM16A. Sci Rep (resubmitted). https://doi.org/10.1096/fj.201600797RR

2. Botelho HM, Uliyakina I, Awatade NT, Proenca MC, Tischer C, Sirianant L, Kunzelmann K, Pepperkok R, Amaral MD (2015) Protein traffic disorders: an effective high-throughput fluorescence microscopy pipeline for drug discovery. Sci Rep 5:9038

3. Bradbury NA, Jilling T, Gabor B, Sorscher EJ, Bridges RJ, Kirk KL (1992) Regulation of plasma membrane recycling by CFTR. Science 256:530–531

4. Brunner JD, Lim NK, Schenck S, Duerst A, Dutzler R (2014) X-ray structure of a calcium-activated TMEM16 lipid scramblase. Nature 516:207–212

5. Brunner JD, Schenck S, Dutzler R (2016) Structural basis for phospholipid scrambling in the TMEM16 family. Curr Opin Struct Biol 39:61–70. https://doi.org/10.1016/j.sbi.2016.05.020

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