Nitrosative stress, protein tyrosine nitration, PARP activation and NAD depletion in the kidneys of rats after single dose of cyclophosphamide
Author:
Publisher
Springer Science and Business Media LLC
Subject
Physiology (medical),Nephrology,Physiology
Link
http://link.springer.com/content/pdf/10.1007/s10157-009-0160-z.pdf
Reference37 articles.
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2. Fleming RA. An overview of cyclophosphamide and ifosfamide pharmacology. Pharmacotherapy. 1997;17:146–54 S.
3. Trefler J, Matyska-Piekarska E, Łackiacki JK. Cyclophosphamide in the therapy of rheumatoid arthritis and its complications (in Polish). Pol Merkur Lekarski. 2007;22:566–70.
4. Sugumar E, Kanakasabapathy I, Abraham P. Normal plasma creatinine level despite histological evidence of damage and increased oxidative stress in the kidneys of cyclophosphamide treated rats. Clin Chim Acta. 2007;376:244–5.
5. Ghosh S, Ghosh D, Chattopadhyay S, Debnath J. Effect of ascorbic acid supplementation on liver and kidney toxicity in cyclophosphamide-treated female albino rats. J Toxicol Sci. 1999;24:141–4.
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