Endothelial extracellular vesicles induce acute lung injury via follistatin-like protein 1-Reference-Cited by-同舟云学术

Endothelial extracellular vesicles induce acute lung injury via follistatin-like protein 1

Published:2023-05-17 Issue: Volume: Page:
ISSN:1674-7305
Container-title:Science China Life Sciences
language:en
Short-container-title:Sci. China Life Sci.

Author:

Yuan Hao-Xiang,Chen Ya-Ting,Li Yu-Quan,Wang Yan-Sheng,Ou Zhi-Jun,Li Yan,Gao Jian-Jun,Deng Meng-Jie,Song Yuan-Kai,Fu Li,Ci Hong-Bo,Chang Feng-Jun,Cao Yang,Jian Yu-Peng,Kang Bi-Ang,Mo Zhi-Wei,Ning Da-Sheng,Peng Yue-Ming,Liu Ze-Long,Liu Xiao-Jun,Xu Ying-Qi,Xu Jun,Ou Jing-Song

Publisher

Springer Science and Business Media LLC

Subject

General Agricultural and Biological Sciences,General Environmental Science,General Biochemistry, Genetics and Molecular Biology

Link

https://link.springer.com/content/pdf/10.1007/s11427-022-2328-x.pdf

Reference51 articles.

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2. Antunes, M.A., Braga, C.L., Oliveira, T.B., Kitoko, J.Z., Castro, L.L., Xisto, D.G., Coelho, M.S., Rocha, N., Silva-Aguiar, R.P., Caruso-Neves, C., et al. (2021). Mesenchymal stromal cells from emphysematous donors and their extracellular vesicles are unable to reverse cardiorespiratory dysfunction in experimental severe emphysema. Front Cell Dev Biol 9, 661385.

3. Benedikter, B.J., Bouwman, F.G., Vajen, T., Heinzmann, A.C.A., Grauls, G., Mariman, E.C., Wouters, E.F.M., Savelkoul, P.H., Lopez-Iglesias, C., Koenen, R.R., et al. (2017). Ultrafiltration combined with size exclusion chromatography efficiently isolates extracellular vesicles from cell culture media for compositional and functional studies. Sci Rep 7, 15297.

4. Buesing, K.L., Densmore, J.C., Kaul, S., Pritchard Jr., K.A., Jarzembowski, J.A., Gourlay, D.M., and Oldham, K.T. (2011). Endothelial microparticles induce inflammation in acute lung injury. J Surg Res 166, 32–39.

5. Bugueno, I.M., Zobairi El-Ghazouani, F., Batool, F., El Itawi, H., Anglès-Cano, E., Benkirane-Jessel, N., Toti, F., and Huck, O. (2020). Porphyromonas gingivalis triggers the shedding of inflammatory endothelial microvesicles that act as autocrine effectors of endothelial dysfunction. Sci Rep 10, 1778.

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