Empagliflozin attenuates doxorubicin-impaired cardiac contractility by suppressing reactive oxygen species in isolated myocytes

Author:

Lin Rong,Peng Xiaodong,Li Yukun,Wang Xuesi,Liu Xinmeng,Jia Xingze,Zhang Chengjun,Liu Nian,Dong Jianzeng

Funder

National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Reference41 articles.

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2. Buzdar AU, Marcus C, Smith TL, Blumenschein GR (1985) Early and delayed clinical cardiotoxicity of doxorubicin. Cancer 55:2761–2765. https://doi.org/10.1002/1097-0142(19850615)55:12%3c2761::aid-cncr2820551206%3e3.0.co;2-p

3. Zamorano JL, Lancellotti P, Rodriguez Muñoz D, Aboyans V, Asteggiano R, Galderisi M, Habib G, Lenihan DJ, Lip GYH, Lyon AR, Lopez Fernandez T, Mohty D, Piepoli MF, Tamargo J, Torbicki A, Suter TM (2016) 2016 ESC position paper on cancer treatments and cardiovascular toxicity developed under the auspices of the ESC Committee for Practice guidelines: the task force for cancer treatments and cardiovascular toxicity of the European Society of Cardiology (ESC). Eur Heart J 37:2768–2801. https://doi.org/10.1093/eurheartj/ehw211

4. Middleman E, Luce J, Frei E 3rd (1971) Clinical trials with adriamycin. Cancer 28:844–850. https://doi.org/10.1002/1097-0142(1971)28:4%3c844::aid-cncr2820280407%3e3.0.co;2-9

5. Felker GM, Thompson RE, Hare JM, Hruban RH, Clemetson DE, Howard DL, Baughman KL, Kasper EK (2000) Underlying causes and long-term survival in patients with initially unexplained cardiomyopathy. N Engl J Med 342:1077–1084. https://doi.org/10.1056/nejm200004133421502

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