Hereditary hemochromatosis with homozygous C282Y HFE mutation: possible clinical model to assess effects of elevated reactive oxygen species on the development of cardiovascular disease
Author:
Funder
National Heart, Lung, and Blood Institute
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s11010-023-04726-y.pdf
Reference119 articles.
1. Pietrangelo A (2004) Hereditary hemochromatosis–a new look at an old disease. N Engl J Med 350(23):2383–2397. https://doi.org/10.1056/NEJMra031573
2. Gujja P, Rosing DR, Tripodi DJ, Shizukuda Y (2010) Iron overload cardiomyopathy: better understanding of an increasing disorder. J Am Coll Cardiol 56(13):1001–1012. https://doi.org/10.1016/j.jacc.2010.03.083
3. Fleming RE, Ponka P (2012) Iron overload in human disease. N Engl J Med 366(4):348–359. https://doi.org/10.1056/NEJMra1004967
4. Shizukuda Y, Bolan CD, Nguyen TT, Botello G, Tripodi DJ, Yau YY et al (2007) Oxidative stress in asymptomatic subjects with hereditary hemochromatosis. Am J Hematol 82(3):249–250. https://doi.org/10.1002/ajh.20743
5. Shizukuda Y, Lingamaneni P, Rosing DR (2021) Incidence of symptomatic arrhythmias and utilization of arrhythmia testing in 10-year follow up of cardiac asymptomatic hereditary hemochromatosis subjects. Am J Cardiol 157:153–154. https://doi.org/10.1016/j.amjcard.2021.07.031
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