Myocardial infarction in mice alters sarcomeric function via post-translational protein modification-Reference-Cited by-同舟云学术

Myocardial infarction in mice alters sarcomeric function via post-translational protein modification

Published:2011-12-08 Issue:1-2 Volume:363 Page:203-215
ISSN:0300-8177
Container-title:Molecular and Cellular Biochemistry
language:en
Short-container-title:Mol Cell Biochem

Author:

Avner Benjamin S.,Shioura Krystyna M.,Scruggs Sarah B.,Grachoff Milana,Geenen David L.,Helseth Donald L.,Farjah Mariam,Goldspink Paul H.,John Solaro R.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,General Medicine

Link

http://link.springer.com/content/pdf/10.1007/s11010-011-1172-z.pdf

Reference45 articles.

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2. Layland J, Solaro RJ, Shah AM (2005) Regulation of cardiac contractile function by troponin I phosphorylation. Cardiovasc Res 66:12–21

3. Vahebi S, Ota A, Li M, Warren CM, de Tombe PP, Wang Y, Solaro RJ (2007) p38-MAPK induced dephosphorylation of alpha-tropomyosin is associated with depression of myocardial sarcomeric tension and ATPase activity. Circ Res 100:408–415

4. Seidman JG, Seidman C (2001) The genetic basis for cardiomyopathy: from mutation identification to mechanistic paradigms. Cell 104:557–567

5. Jacques AM, Copeland O, Messer AE, Gallon CE, King K, McKenna WJ et al (2008) Myosin binding protein C phosphorylation in normal, hypertrophic and failing human heart muscle. J Mol Cell Cardiol 45:209–216

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