Polymorphonuclear neutrophils promote endothelial apoptosis by enhancing adhesion upon stimulation by intermittent hypoxia
Author:
Funder
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
Neurology (clinical),Otorhinolaryngology
Link
https://link.springer.com/content/pdf/10.1007/s11325-021-02503-z.pdf
Reference41 articles.
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2. Garvey JF, Taylor CT, McNicholas WT (2009) Cardiovascular disease in obstructive sleep apnoea syndrome: the role of intermittent hypoxia and inflammation. Eur Respir J 33(5):1195–1205. https://doi.org/10.1183/09031936.00111208
3. Arnaud C, Bochaton T, Pepin JL, Belaidi E (2020) Obstructive sleep apnoea and cardiovascular consequences: pathophysiological mechanisms. Arch Cardiovasc Dis 113(5):350–358. https://doi.org/10.1016/j.acvd.2020.01.003
4. Javaheri S, Barbe F, Campos-Rodriguez F, Dempsey JA, Khayat R, Javaheri S, Malhotra A, Martinez-Garcia MA, Mehra R, Pack AI, Polotsky VY, Redline S, Somers VK (2017) Sleep apnea: types, mechanisms, and clinical cardiovascular consequences. J Am Coll Cardiol 69(7):841–858. https://doi.org/10.1016/j.jacc.2016.11.069
5. Badran M, Golbidi S, Devlin A, Ayas N, Laher I (2014) Chronic intermittent hypoxia causes endothelial dysfunction in a mouse model of diet-induced obesity. Sleep Med 15(5):596–602. https://doi.org/10.1016/j.sleep.2014.01.013
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