High Prion and PrPSc Levels but Delayed Onset of Disease in Scrapie-Inoculated Mice Heterozygous for a Disrupted PrP Gene-Reference-Cited by-同舟云学术

High Prion and PrPSc Levels but Delayed Onset of Disease in Scrapie-Inoculated Mice Heterozygous for a Disrupted PrP Gene

Published:1994-11 Issue:1 Volume:1 Page:19-30
ISSN:1076-1551
Container-title:Molecular Medicine
language:en
Short-container-title:Mol Med

Author:

Büeler Hansruedi,Raeber Alex,Sailer Andreas,Fischer Marek,Aguzzi Adriano,Weissmann Charles

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Genetics,Molecular Biology,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1007/BF03403528.pdf

Reference38 articles.

1. Prusiner SB. (1982) Novel proteinaceous infectious particles cause scrapie. Science 216: 136–144.

2. Prusiner SB. (1989) Scrapie prions. Annu. Rev. Microbiol. 43: 345–374.

3. Prusiner SB, Scott M, Foster D, et al. (1990) Transgenetic studies implicate interactions between homologous PrP isoforms in scrapie prion replication. Cell 63: 673–686.

4. Prusiner SB. (1993) Transgenetic investigations of prion diseases of humans and animals. Philos. Trans. R. Soc. Lond. Biol. 339: 239–254.

5. Weissmann C, Büeler H, Fischer M, Aguet M. (1993) Role of the PrP gene in transmissible spongiform encephalopathies. In: Zinkernagel RM, Stauffacher W (eds). Viruses and Virus-like Agents in Disease. Karger, Basel, pp. 164–175.

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