Tripartite motif-containing protein 16 protects against myocardial ischemia/reperfusion injury by affecting the Keap1/Nrf2 axis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Histology,Pathology and Forensic Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00441-021-03518-4.pdf
Reference32 articles.
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3. Bell JL, Malyukova A, Kavallaris M, Marshall GM, Cheung BB (2013) TRIM16 inhibits neuroblastoma cell proliferation through cell cycle regulation and dynamic nuclear localization. Cell Cycle 12:889–898
4. Cheung BB, Bell J, Raif A, Bohlken A, Yan J, Roediger B, Poljak A, Smith S, Lee M, Thomas WD, Kavallaris M, Norris M, Haber M, Liu HL, Zajchowski D, Marshall GM (2006) The estrogen-responsive B box protein is a novel regulator of the retinoid signal. J Biol Chem 281:18246–18256
5. Frangogiannis NG (2015) Pathophysiology of Myocardial Infarction Compr Physiol 5:1841–1875
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1. Upregulation of TRIM16 mitigates doxorubicin-induced cardiotoxicity by modulating TAK1 and YAP/Nrf2 pathways in mice;Biochemical Pharmacology;2024-02
2. TRIM16 exerts protective function on myocardial ischemia/reperfusion injury through reducing pyroptosis and inflammation via NLRP3 signaling;Biochemical and Biophysical Research Communications;2022-12
3. Targeting Nrf2 in ischemia-reperfusion alleviation: From signaling networks to therapeutic targeting;Life Sciences;2022-07
4. The E3 Ligase TRIM16 Is a Key Suppressor of Pathological Cardiac Hypertrophy;Circulation Research;2022-05-13
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