Acute and subacute oral administration of mycotoxin deoxynivalenol exacerbates the pro-inflammatory and pro-pruritic responses in a mouse model of allergic dermatitis
Author:
Funder
Agriculture, Forestry and Fisheries Research Council
Tojuro Iijima Foundation for Food Science and Technology
Publisher
Springer Science and Business Media LLC
Subject
Health, Toxicology and Mutagenesis,Toxicology,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s00204-020-02875-3.pdf
Reference39 articles.
1. Ahn K, Kim BE, Kim J, Leung DY (2020) Recent advances in atopic dermatitis. Curr Opin Immunol 66:14–21. https://doi.org/10.1016/j.coi.2020.02.007
2. Amuzie CJ, Shinozuka J, Pestka JJ (2009) Induction of suppressors of cytokine signaling by the trichothecene deoxynivalenol in the mouse. Toxicol Sci 111(2):277–287. https://doi.org/10.1093/toxsci/kfp150
3. Ashikaga T, Yoshida Y, Hirota M et al (2006) Development of an in vitro skin sensitization test using human cell lines: the human Cell Line Activation Test (h-CLAT). I. Optimization of the h-CLAT protocol. Toxicol In Vitro 20(5):767–773. https://doi.org/10.1016/j.tiv.2005.10.012
4. Bell BD, Kitajima M, Larson RP et al (2013) The transcription factor STAT5 is critical in dendritic cells for the development of TH2 but not TH1 responses. Nat Immunol 14(4):364–371. https://doi.org/10.1038/ni.2541
5. Bol-Schoenmakers M, Braber S, Akbari P et al (2016) The mycotoxin deoxynivalenol facilitates allergic sensitization to whey in mice. Mucosal Immunol 9(6):1477–1486. https://doi.org/10.1038/mi.2016.13
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