Increased β-Cell Apoptosis Prevents Adaptive Increase in β-Cell Mass in Mouse Model of Type 2 Diabetes
Author:
Affiliation:
1. Division of Endocrinology and Diabetes, Keck School of Medicine, University of Southern California, Los Angeles, California
2. Karolinska Institutet, Stockholm, Sweden
3. Pfizer Global Research and Development, Pfizer, Groton, Connecticut
Abstract
Publisher
American Diabetes Association
Subject
Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
https://journals.org/diabetes/diabetes/article-pdf/52/9/2304/650281/db0903002304.pdf
Reference54 articles.
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3. Butler AE, Janson J, Bonner-Weir S, Ritzel R, Rizza RA, Butler PC: β-Cell deficit and increased β-cell apoptosis in humans with type-2 diabetes mellitus. Diabetes 52:102–110,2003
4. Tyrberg B, Ustinov J, Otonkoski T, Andersson A: Stimulated endocrine cell proliferation and differentiation in transplanted human pancreatic islets: effects of the ob gene and compensatory growth of the implantation organ. Diabetes 50:301–307,2001
5. Kloppel G, Lohr M, Habich K, Oberholzer M, Heitz PU: Islet pathology and the pathogenesis of type 1 and type 2 diabetes mellitus revisited. Surv Synth Pathol Res 4:110–125,1985
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